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DESCRIPTION
Colorblindness is a general term for the inability to see colors as most people see them. Specialized cells in the retina known as cones, which are vital for translating light waves into a perception of color, may be completely or partially lacking, or may not function properly, resulting in color blindness. There are different types and varying severity of this condition. Most colorblind people confuse certain colors (they may not be able to tell red from green, for example); in rare cases, an individual may see no color at all. Some can distinguish colors only in certain lights.
ColorTesting.com: Colorblind Homepages
This website defines being color blind and provides information about the different types of color blindness and pediatric Colorblindness testing for small children. Very informative.
There are certain diseases, including pernicious anemia and sickle cell disease, eye, nerve or brain damage, as well as number of different medications and exposure to certain chemicals, that can cause disturbances in color vision.
Color blindness is usually classified as a disability, however, in certain selected situations, color blind people have an advantage over people with normal vision. There are some studies which have found that color blind people are better at penetrating certain camouflages. Monochromats may have a minor advantage in dark vision, but only in the first 5-1/2 minutes of dark adaptation.
Because few people are tested for color vision, color blindness is probably an under diagnosed condition, especially among women. Colorblindness is typically a genetic condition, and it is much more common in men than in women. The gene for color blindness is located on the X sex chromosome (X-Linked) and is passed from mother to son. Approximately 1 in 12 men has at least some color perception problems. In most cases, color blindness is present from birth, although acquired deficiencies stem from injury, disease, or the aging process. Although not called "color blindness," when people age, their corneas typically turn yellowish, severely hampering their ability to see violet and blue colors. The dimming of vision caused by cataracts, can diminish a person's ability to distinguish colors later in life.
The normal human retina contains two kinds of light cells: the rod cells (active in low light) and the cone cells (active in normal daylight). Normally, there are three kinds of cones, each containing a different pigment. The cones are activated when the pigments absorb light. The absorption spectra of the cones differ; one is maximally sensitive to short wavelengths, one to medium wavelengths, and the third to long wavelengths (their peak sensitivities are in the blue, yellowish-green, and yellow regions of the spectrum, respectively). The absorption spectra of all three systems cover much of the visible spectrum, so it is not entirely accurate to refer to them as "blue", "green" and "red" receptors, especially because the "red" receptor actually has its peak sensitivity in the yellow. The sensitivity of normal color vision actually depends on the overlap between the absorption spectra of the three systems: different colors are recognized when the different types of cone are stimulated to different extents. Red light, for example, stimulates the long wavelength cones much more than either of the others, and reducing wavelength causes the other two cone systems to be increasingly stimulated, causing a gradual change in hue.
CAUSES OF COLOR BLINDNESS
GENETICS
Color blindness can be inherited genetically. Some people believe, incorrectly, that it is only ever inherited from mutations on the X chromosome. Since the mapping of the human genome there have been many causative mutations discovered. Mutations capable of causing color blindness originate from at least 19 different chromosomes and many different genes.
These are some of the inherited diseases known to cause color blindness:
- Cone Dystrophy
- Cone-Rod Dystrophy
- Achromatopsia (aka Rod Monochromatism, aka Stationery Cone Dystrophy, aka Cone Dysfunction Syndrome)
- Blue Cone Monochromatism
- Retinitis Pigmentosa (initially affects rods but can later progress to cones and therefore color blindness),
- Diabetes
- Age-Related Macula Degeneration
- Retinoblastoma
- Lebers Congenital Amorosis
Inherited color blindness can be congenital (color blind from birth) or it can commence in childhood or adulthood. Depending on the mutation, it can be stationery (remain the same throughout a person's lifetime) or progressive. Because of the nature of progressive phenotypes deteriorating the retina and other parts of the eye, certain forms of color blindness can progress to legal blindness (an a acuity of 6/60 or worse) and often leave a person with complete blindness.
Color blindness always pertains to the cone photoreceptors in our retina as the cones are capable of detecting the color frequencies of light we perceive. There are 3 types of cones, each responsible for detecting either red, green or blue.
About 2 percent of females and 8 percent of males are color blind. The reason males are at a greater risk of inheriting an X linked mutation is because males only have one X chromosome (XY), and females have two (XX). In color blindness caused by mutations on the X chromosome there is a 50% chance of male offspring being affected and a 50% chance of female offspring being carriers. Nature usually deals with mutated genes by expressing the healthy copy in offspring. Males only receive one copy and are therefore more vulnerable to mutations on the X chromosome being passed to them by their mothers. If the X chromosome passed to a male carries a color blindness causing mutation then the male will be color blind because there is no chance of another X chromosome silencing the mutation. If one of the X chromosomes of a female carries the gene for color blindness, generally the other will not, so there is a dominant gene to take the place of the recessive one.
NON-GENETIC CAUSES
Shaken Baby Syndrome can cause damage to the retina and brain damage and therefore cause color blindness.
Accidents and other trauma to the retina and brain.
UV damage (not wearing appropriate protection). Most UV damage is caused during childhood and this form of retinal degeneration is the leading cause of blindness in the world. Damage often presents later in life.
TYPES OF COLOR BLINDNESS
There are many types of color blindness. The most common are red-green hereditary (genetic) photoreceptor disorders, but it is also possible to acquire color blindness through damage to the retina, optic nerve, or higher brain areas. Color vision deficiencies can be classified as acquired or inherited.
ACQUIRED: Damage to higher brain areas implicated in color processing include the parvocellular pathway of the lateral geniculate nucleus of the thalamus, and visual area V4 of the visual cortex. Acquired color blindness is generally unlike the more typical genetic disorders. For example, it is possible to acquire color blindness only in a portion of the visual field but maintain normal color vision elsewhere. Some forms of acquired color blindness are reversible. Transient color blindness also occurs (very rarely) in the aura of some migraine sufferers.
INHERITED: There are three types of inherited or congenital color vision deficiencies: monochromacy, dichromacy, and anomalous trichromacy.MONOCHROMACY
Monochromacy is also known as "total color blindness", is the lack of ability to distinguish any colors and perceive only variations in brightness. It is caused by cone defect or absence. Monochromacy occurs when two or all three of the cone pigments are missing and color and lightness vision is reduced to one dimension.
Rod monochromacy (Achromatopsia) is a rare, non-progressive inability to distinguish any colors as a result of absent or non-functioning retinal cone cells, so that in addition to the absence of color discrimination, vision in light of normal intensity is difficult. It is associated with light sensitivity (Photophobia), involuntary eye oscillations (nystagmus), and poor vision. While normally rare, Achromatopsia is very common on the island of Pingelap, a part of the Pohnpei state, Federated States of Micronesia, where it is called maskun: about 1/12 of the population there has it. The island was devastated by a storm in the 18th century, and one of the few male survivors carried a gene for Achromatopsia; the population is now several thousand, of whom about 30 percent carry this gene.
Cone monochromacy is a rare, total color blindness that is accompanied by relatively normal vision, electoretinogram, and electrooculogram. It is a condition of having both rods and cones, but only a single kind of cone. A cone monochromat can have good pattern vision at normal daylight levels, but will not be able to distinguish hues. Blue cone monochromacy (X chromosome) is caused by a complete absence of L- and M-cones. It is encoded at the same place as red-green color blindness on the X chromosome. Peak spectral sensitivities are in the blue region of the visible spectrum (near 440 nm). They generally show nystagmus ("jiggling eyes"), photophobia (light sensitivity), reduced visual acuity, and myopia (nearsightedness). Visual acuity usually falls to the 20/50 to 20/400 range.
DICHROMACY
Dichromacy is a moderately severe color vision defect in which one of the three basic color mechanisms is absent or not functioning. It is hereditary and sex-linked, affecting predominantly males. Dichromacy occurs when one of the cone pigments is missing and color is reduced to two dimensions. Protanopes, deuteranopes, and tritanopes are dichromats. They can match any color they see with some mixture of just two spectral lights (whereas normally humans are trichromats and require three lights). These individuals normally know they have a color vision problem and it can affect their lives on a daily basis. Protanopes and deuteranopes see no perceptible difference between red, orange, yellow, and green. All these colors that seem so different to the normal viewer appear to be the same color for this 2 percent of the population.
Protanopia is a severe type of color vision deficiency caused by the complete absence of red retinal photoreceptors. It is a form of dichromatism in which red appears dark. It is hereditary, sex-linked, and present in 1 percent of all males. Lacking the long-wavelength sensitive retinal cones, those with this condition are unable to distinguish between colors in the green-yellow-red section of the spectrum. They have a neutral point at a greenish wavelength around 492 nm - they cannot discriminate light of this wavelength from white. For the protanope, the brightness of red, orange, and yellow is much reduced compared to normal. This dimming can be so pronounced that reds may be confused with black or dark gray, and red traffic lights may appear to be extinguished. They may learn to distinguish reds from yellows and from greens primarily on the basis of their apparent brightness or lightness, not on any perceptible hue difference. Violet, lavender, and purple are indistinguishable from various shades of blue because their reddish components are so dimmed as to be invisible. For example, Pink flowers, reflecting both red light and blue light, may appear just blue to the protanope. Very few people have been found who have one normal eye and one protanopic eye. These unilateral dichromats report that with only their protanopic eye open, they see wavelengths below the neutral point as blue and those above it as yellow. This is a rare form of color blindness.
Deuteranopia is a color vision deficiency in which the green retinal photoreceptors are absent, moderately affecting red-green hue discrimination. It is a form of dichromatism in which there are only two cone pigments present. It is likewise hereditary, sex-linked, and present in 1 percent of all males. Lacking the medium-wavelength cones, those affected are again unable to distinguish between colors in the green-yellow-red section of the spectrum. Their neutral point is at a slightly longer wavelength, 498 nm. The deuteranope suffers the same hue discrimination problems as the protanope, but without the abnormal dimming. The names red, orange, yellow, and green really mean very little to him aside from being different names that every one else around him seems to be able to agree on. Similarly, violet, lavender, purple, and blue, seem to be too many names to use logically for hues that all look alike to him. This is one of the rarer forms of color blindness, also known as Daltonism after John Dalton. (Dalton's diagnosis was confirmed as deuteranopia in 1995, some 150 years after his death, by DNA analysis of his preserved eyeball.) Deuteranopic unilateral dichromats report that with only their deuteranopic eye open, they see wavelengths below the neutral point as blue and those above it as yellow.
Tritanopia is an exceedingly rare color vision disturbance in which there are only two cone pigments present and a total absence of blue retinal receptors. Lacking the short-wavelength cones, those affected are unable to distinguish between the colors in the blue-yellow section of the spectrum. This form of color blindness is not sex-linked and affects less than 1 percent of males and females.
ANOMALOUS TRICHROMACY
Anomalous trichromacy is a common type of inherited color vision deficiency, occurring when one of the three cone pigments is altered in its spectral sensitivity. This results in an impairment, rather than loss, of trichromacy (normal three-dimensional color vision). Those with protanomaly, deuteranomaly, or tritanomaly are trichromats, but the color matches they make differ from the normal. They are called anomalous trichromats. In order to match a given spectral yellow light, protanomalous observers need more red light in a red/green mixture than a normal observer, and deuteranomalous observers need more green. From a practical stand point though, many protanomalous and deuteranomalous people breeze through life with very little difficulty doing tasks that require normal color vision. Some may not even be aware that their color perception is in any way different from normal. The only problem they have is passing a color vision test.
Protanomaly and deuteranomaly can be readily observed using an instrument called an anomaloscope, which mixes spectral red and green lights in variable proportions, for comparison with a fixed spectral yellow. If this is done in front of a large audience of men, as the proportion of red is increased from a low value, first a small proportion of people will declare a match, while most of the audience sees the mixed light as greenish. These are the deuteranomalous observers. Next, as more red is added the majority will say that a match has been achieved. Finally, as yet more red is added, the remaining, protanomalous, observers will declare a match at a point where everyone else is seeing the mixed light as definitely reddish.
Protanomaly is a mild color vision defect in which an altered spectral sensitivity of red retinal receptors (closer to green receptor response) results in poor red-green hue discrimination. It is hereditary, sex-linked, and present in 1 percent of all males and 0.01 percent of females. It is often passed from mother to child. Having a mutated form of the long-wavelength (red) pigment, whose peak sensitivity is at a shorter wavelength than in the normal retina, protanomalous individuals are less sensitive to red light than normal. This means that they are less able to discriminate colors, and they do not see mixed lights as having the same colors as normal observers. They also suffer from a darkening of the red end of the spectrum. This causes reds to reduce in intensity to the point where they can be mistaken for black. Protanomaly is a fairly rare form of color blindness. Both protanomaly and deuteranomaly are carried on the X chromosome.
Deuteranomaly is caused by a similar shift in the green retinal receptors, is by far the most common type of color vision deficiency, mildly affecting red-green hue discrimination in 6 percent of all males and 0.4 percent of females. It is hereditary and sex-linked. Having a mutated form of the medium-wavelength (green) pigment. The medium-wavelength pigment is shifted towards the red end of the spectrum resulting in a reduction in sensitivity to the green area of the spectrum. Unlike protanomaly the intensity of colors is unchanged. The deuteranomalous person is considered "green weak". For example, in the evening, dark green cars appear to be black to Deuteranomalous people. Similar to the protanomates, deuteranomates are poor at discriminating small differences in hues in the red, orange, yellow, green region of the spectrum. They make errors in the naming of hues in this region because the hues appear somewhat shifted towards red. One very important difference between deuteranomalous individuals and protanomalous individuals is deuteranomalous individuals do not have the loss of "brightness" problem.
Tritanomaly is a rare, hereditary color vision deficiency affecting blue-yellow hue discrimination and is equally rare for males and females. Having a mutated form of the short-wavelength (blue) pigment. The short-wavelength pigment is shifted towards the green area of the spectrum. This is the rarest form of anomalous trichromacy color blindness. Unlike the other anomalous trichromasy color deficiencies, the mutation for this color blindness is carried on chromosome 7. Therefore it is equally prevalent in both male and female populations. The OMIM gene code for this mutation is 304000 "Colorblindness, Partial Tritanomaly."
DIAGNOSIS OF COLOR BLINDNESS
The Ishihara color test, which consists of a series of pictures of colored spots, is the test most often used to diagnose red-green color deficiencies. A figure (usually one or more Arabic digits) is embedded in the picture as a number of spots in a slightly different color, and can be seen with normal color vision, but not with a particular color defect. The full set of tests has a variety of figure/background color combinations, and enable diagnosis of which particular visual defect is present. The anomaloscope, described above, is also used in diagnosing anomalous trichromacy.
However, the Ishihara color test is criticized for containing only numerals and thus not being useful for young children, who have not yet learned to use numerals. It is important to identify these problems as soon as possible and explain them to the children to prevent possible problems and psychological traumas. For this reason, alternative color vision tests were developed using only symbols (square, circle, car).
Most clinical tests are designed to be fast, simple, and effective at identifying broad categories of color blindness. In academic studies of color blindness, on the other hand, there is more interest in developing flexible tests to collect thorough datasets, identify copunctal points, and measure just noticeable differences.
MANAGING COLOR BLINDNESS
There is generally no treatment to cure color deficiencies. However, certain types of tinted filters and contact lenses may help an individual to distinguish different colors better. Optometrists can supply a singular red-tint contact lens to wear in the dominant eye. This may enable the wearer to pass color blindness tests for certain occupations. The effect of wearing such a device is akin to wearing red/blue 3D glasses and can take some getting used to as certain wavelengths can "jump" out and be overly represented. Additionally, computer software has been developed to assist those with visual color difficulties.
The Gnome Desktop provides colorblind accessibility using the gnome-mag and the libcolorblind software. Using a gnome applet, the user may switch a color filter on and off choosing from a set of possible color transformations that will displace the colors in order to disambiguate the colors. The software enables, for instance, a color blind person to see the numbers in the ishihara test.
The National Eye Institute is doing research into treating/curing color blindness, and it is now required to donate 5 percent of its resources to this cause under instruction of the National Institutes of Health.
GRAPHIC DESIGNS & COLOR BLINDNESS
Color codes present particular problems for color blind people as they are often difficult or impossible for color blind people to understand. Good graphic design avoids using color coding or color contrasts alone to express information, as this not only helps color blind people, but also aids understanding by normally sighted people. The use of Cascading Style Sheets on the world wide web allows pages to be given an alternative color scheme for color-blind readers. This color scheme generator helps a graphic designer see color schemes as seen by eight types of color blindness. For an example of a map that could present a significant problem to a color blind reader. The typical red-green color blind reader will find the green sections of the map nearly indistinguishable from the orange, rendering the graphic unreadable.
Designers should take into account that color-blindness is highly sensitive to differences in material. For example, a red-green colorblind person who is incapable of distinguishing colors on a map printed on paper may have no such difficulty when viewing the map on a computer screen or television. In addition, some color blind people find it easier to distinguish problem colors on artificial materials, such as plastic or in acrylic paints, than on natural materials, such as paper or wood. Thirdly, for some color blind people, color can only be distinguished if there is a sufficient "mass" of color: thin lines might appear black while a thicker line of the same color can be perceived as having color.
When the need to process visual information as rapidly as possible arises, for example in a train or aircraft crash, the visual system may operate only in shades of grey, with the extra information load in adding color being dropped. This is an important possibility to consider when designing, for example, emergency brake handles or emergency phones. Due to this inability to recognize colors such as red and green, some countries (Singapore prior to the 1990s or Romania even to the present day) have refused to grant individuals with color blindness driving licenses. In Romania there is an undergoing effort to remove the legal restrictions that prohibit its colorblind citizens from getting drivers' licenses.
CLARIFYING MISCONCEPTIONS ABOUT COLOR BLINDNESS
Color blindness is not the swapping of colors in the observer's eyes. Grass is never red, and stop signs are never green. The color impaired do not learn to call red "green" and vice versa. However, dichromats often confuse red and green items. For example, they may find it difficult to distinguish a Braeburn apple from a Granny Smith apple and in some cases, the red and green of a traffic light without other clues (shape or location). This is demonstrated in this simulation of the two types of apple as viewed by a trichromat or by a dichromat.
Anomalous Trichromats are often able to readily spot camouflage clothing, netting, and paint that has been designed for individuals with color-normal vision. They tend to learn to see texture and shape. This lets them see through some camouflage patterns. In the apple example, above, they will see the clear difference because the surface pattern is different.
Traffic light colors are confusing to some dichromats: there is insufficient apparent difference between the red and amber and sodium street lamps and the green can be confused with a grubby white lamp. This is a risk factor on a high-speed undulating road where angular cues can't be used. British Rail color lamp signals use more easily identifiable colors: the red is really blood red, the amber is quite yellow and the green is a bluish color.
Color blindness almost never means complete monochromatism. In almost all cases, color blind people retain blue-yellow discrimination, and most color blind individuals are anomalous trichromats rather than complete dichromats. In practice this means that they often retain a limited discrimination along the red-green axis of color space although their ability to separate colors in this dimension is severely reduced.
It should also be noted that even though some people are unable to see some or maybe even any of the numbers in (e.g. red-green) color blindness tests, they might still be able to tell the difference between the colors in their everyday lives.
Information obtained in part from: Wikipedia - Color Blindness, Click on link to see full article, graphics and citations.
HOLISTIC RECOMMENDATIONS & NUTRITION
NUTRITIONAL SUPPLEMENT RECOMMENDATIONS
The following nutrients are important for healing once appropriate local treatment has been administered. Unless otherwise specified, the following recommended doses are for those over the age of 18. For a child between 12 and 17 years old, reduce the dose to 3/4 the recommended amount. For a child between 6 and 12 years old, use 1/2 the recommended dose, and for a child under 6, use 1/4 the recommended amount.
NUTRIENTS Supplement Suggested Dosage Comments Vitamin A 25,000-50,000 IU daily. If you are pregnant, do not exceed 10,000 IU daily. May be helpful because it is essential for proper functioning of the cones in the retina. Also improves night blindness. Use emulsion form, which is safe at higher doses than capsules.
Vitamin A, 10,000 IU, 100% Natural, Nature's Way, 100 SoftgelsPlus
Natural Beta-Carotene
Or
Carotenoid ComplexAs directed on label. May be helpful because it is essential for proper functioning of the cones in the retina. Also improves night blindness.
Beta Carotene (Natural Dunaliella Salina), Nature's Way, 100% Natural, 25,000 IU, 100 Softgels,
Multi-Carotene Antioxidant, Nature's Way, 60 SoftgelsPlus
LuteinAs directed on label. Lutein can help protect the retina from harmful ultraviolet (UV) light and free radicals.
Lutein Supplement, Nature's Way, 20 mg, 60 Softgels,
Lutein Esters, 20 mg, 60 SoftgelsVitamin B-12 2,000 mcg daily. Deficiency can lead to yellow-blue color blindness.
Vitamin B-12 Complex Liquid, NOW Foods, 2 fl. oz.,
Vitamin B-12, Nature's Way, 2000 mcg, 100 Sublingual Lozenges,
Vitamin B-12 Liquid Supplement, 50 mcg, With Vitamin B-9 (Folic Acid), 400 mcg, 1 oz.
Vitamin B-12, California Natural, 1000 mcg, 60 Tabs,
Vitamin B-12 LipoSpray, NOW Foods, 2 fl. oz.
COLOR BLINDNESS SUPPLEMENTS & PRODUCTS
Information, supplements and products for color blindness, a general term for the inability to see colors the way most people see them.
Beta Carotene (Natural Dunaliella Salina), 100% Natural, Nature's Way, 25,000 IU, 100 Softgels
Nature's Way Beta Carotene is 100% all natural from Dunaliella Salina.Clear Eye & Sharp Ear, TCM Formula, Shanze Xiaozhi, 60 Caps
Clear Eye & Sharp Ear Chinese Formulas are made of extracts from astragalus root, pueraria root, seashore vitex, common peony root, cimicifuge, phellodendron, licorice, cnidium, Chinese angelica root (Dong Quai) and ginseng.Digestive Enzymes, 750 mg, 100 Caps
Since cooking destroys many of the digestive enzymes in food, taking plant fiber-based digestive enzymes aids in digesting even the heaviest meal.Eye Support Formula With Bilberry, Lutein & Antioxidants, NOW Foods, 60 Caps
NOW "Health Care Provider Recommended" Eye Support Formula eye supplement offers a full range of antioxidant nutrients which may aid in maintaining some visual functions with Lutein and Bilberry.Magnesium Citrate Complex, Nature's Way, 250 mg, 100 Caps
Nature's Way Magnesium Citrate is an advanced chelate from which enhances absorption of magnesium by providing excellent solubility.Neuromins DHA Supplement (Docosahexaenoic Acid), Nature's Way, 100 mg, 60 Softgels
Neuromins brand high quality DHA Supplement is sourced from microalgae and fortified with Vitamin C and E.Once Daily Multi-Vitamin With Iron, All Natural, Lactose Free, Nature's Way, 100 Tabs
Nature's Way Daily Multi Vitamin with Iron provides many of the essential nutrients that modern diets may lack. They are useful for protection, growth and maintenance of body systems as they age.Selenium, 100% Natural, 200 mcg, 100 Caps
Selenium is an essential component of glutathione, the body's most potent natural antioxidant system. It is a popular choice in many antioxidant regimens.Taurine, Free Form, NOW Foods, 500 mg, 100 Caps
Taurine is a conditionally essential amino acid which is not utilized in protein synthesis, but is mainly found free in most tissues, especially throughout the nervous system.Vision Formula With Lutein & Bilberry, Eye Supplement, 60 Caps
Vision formula with Lutein & Bilberry Eye Supplement supports normal eye function by utilizing Lutein, a key carotenoid found in fruits and vegetables, which protects the retina by blocking harmful blue light.Vitamin A, 100% Natural, Nature's Way, 100 Softgels
Nature's Way Vitamin A is 100% natural from fish liver oil. It contains no artificial ingredients or preservatives.Vitamin B-12 Supplements & Products
An assortment of various B-12 products.Zinc Chelate, 100% Natural, Zinc Supplement, Nature's Way, 30 mg, 100 Caps
Nature's Way chelated Zinc supplement is 100% natural chelated with an advanced amino acid complex for superior absorption.
Herbal Remedies: Colorblindness Supplements, Information & Products
Herbal Remedies: Colorblindness Information
Herbal Remedies: Eye Problem Information
RELATED ONLINE LINKS
How do things look to colorblind people?
What is Colorblindness & the Different Types
Colorblindness Tests
Anti-colorblind Tests: Tests For People Who Are Colorblind
Exploring Colorblindness
Colors for the Color Blind
Colorblind Web Page Filter
MoonDragon.org Website has been Color Vision Tested.
TYPES OF EYE PROBLEMS & DISORDERS
Maintaining Healthy Eyes
NOTIFY YOUR HEALTH CARE PROVIDER IF...
You or a family member (such as a child) is having problems distinguishing colors or find colors confusing. This member may need to be tested for color blindness.
You or a family member are having problems with vision and/or you suspect an infection. Call your health care provider immediately if you experience severe eye pain or a sudden change in your vision, such as loss of vision or double vision.
You have any increase of symptoms. You may need frequent changes in your eyeglass prescription. If you have blurred or double vision that develops slowly; are having a problem seeing because of daytime glare or have difficulty driving at night because of glare from headlights, you need to see your health care provider.
You have any unexpected or unusual symptoms. There may be underlying health issues that need to be addressed.
Are having vision problems that are affecting your ability to perform daily activities.
Call your child's health care provider if your baby does not look directly at or respond readily to faces or large, colorful objects by age 2 to 3 months or if your child scowls, squints, or shields his or her eyes more than expected when in sunlight, or light seems to hurt your child's eyes.
You should have your eyesight checked regularly by your health care provider to rule out any problems and to receive a prescription for contacts or eyeglasses, if they are needed. Preserve you vision... it is very important.
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The Complete Guide to Natural Healing
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